Acute bronchoconstriction — definition?
Sudden airway narrowing from inflammation and smooth muscle contraction.
Mast cells — role?
Release mediators causing airway hypersensitivity.
Eosinophils — role?
Contribute to airway inflammation and allergic responses.
Airway inflammation — result?
Swelling, mucus, hyperresponsiveness, bronchoconstriction.
Bronchoconstriction — cause?
Smooth muscle tightening from inflammation.
Hypersensitivity — mechanism?
Exaggerated immune response to allergens.
Asthma — characteristic?
Reversible airway obstruction triggered by irritants.
Diagnosis tools — include?
History, physical, pulmonary tests, allergy skin tests.
Wheezing — sign of?
Airflow limitation in asthma.
Pulmonary function test — purpose?
Assess airflow obstruction severity.
Peak flow monitoring — use?
Quantifies airway narrowing over time.
Accessory muscle use — indicates?
Respiratory distress.
Allergy skin test — purpose?
Identify allergen triggers.
ABG — assesses?
Oxygen and CO2 levels, gas exchange.
Asthma — management?
Inhaled corticosteroids, beta-agonists, trigger avoidance.
Long-term meds — examples?
Fluticasone, montelukast.
Rescue meds — examples?
Albuterol, ipratropium.
COPD — main types?
Emphysema ('pink puffer') and chronic bronchitis ('blue bloater').
Emphysema — features?
Alveolar destruction, hyperinflation, minimal cough.
Chronic bronchitis — features?
Cough, mucus, cyanosis.
Alpha 1 antitrypsin deficiency — role?
Genetic risk factor for emphysema.
Pneumonia — alveolar change?
Filling with fluid, pus, mucus.
Signs of pneumonia?
Fever, chills, tachypnea, crackles, infiltrates.
Diagnosis tools?
X-ray, cultures, ABG, inflammatory markers.
Test your knowledge with 12 questions on Fundamentals of Respiratory and Endocrine Disorders.
1. What is the primary role of mast cells in asthma pathophysiology as described in the source?
2. What is a key feature of pneumonia treatment interventions?
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