Acute bronchoconstriction — definition?
Sudden airway narrowing from inflammation and smooth muscle contraction.
Mast cells — role?
Release mediators causing airway hypersensitivity.
Eosinophils — role?
Contribute to airway inflammation and allergic responses.
Airway inflammation — result?
Swelling, mucus, hyperresponsiveness, bronchoconstriction.
Bronchoconstriction — cause?
Smooth muscle tightening from inflammation.
Hypersensitivity — mechanism?
Exaggerated immune response to allergens.
Asthma — characteristic?
Reversible airway obstruction triggered by irritants.
Diagnosis tools — include?
History, physical, pulmonary tests, allergy skin tests.
Wheezing — sign of?
Airflow limitation in asthma.
Pulmonary function test — purpose?
Assess airflow obstruction severity.
Peak flow monitoring — use?
Quantifies airway narrowing over time.
Accessory muscle use — indicates?
Respiratory distress.
Allergy skin test — purpose?
Identify allergen triggers.
ABG — assesses?
Oxygen and CO2 levels, gas exchange.
Asthma — management?
Inhaled corticosteroids, beta-agonists, trigger avoidance.
Long-term meds — examples?
Fluticasone, montelukast.
Rescue meds — examples?
Albuterol, ipratropium.
COPD — main types?
Emphysema ('pink puffer') and chronic bronchitis ('blue bloater').
Emphysema — features?
Alveolar destruction, hyperinflation, minimal cough.
Chronic bronchitis — features?
Cough, mucus, cyanosis.
Alpha 1 antitrypsin deficiency — role?
Genetic risk factor for emphysema.
Pneumonia — alveolar change?
Filling with fluid, pus, mucus.
Signs of pneumonia?
Fever, chills, tachypnea, crackles, infiltrates.
Diagnosis tools?
X-ray, cultures, ABG, inflammatory markers.
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1. What is the primary role of mast cells in asthma pathophysiology as described in the source?
2. What is a key feature of pneumonia treatment interventions?
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