Rapid Relief: Antacids and PPIs

Study sheet excerpt

Course Outline

  1. Antacids Mechanism
  2. Types of Antacids
  3. Antacids Uses
  4. Antacids Side Effects
  5. PPIs Mechanism
  6. Types of PPIs
  7. PPIs Clinical Uses
  8. PPIs Pharmacokinetics
  9. PPIs Adverse Effects
  10. Antiemetics Mechanisms
  11. Types of Antiemetics
  12. Antiemetics Uses

1. Antacids Mechanism

Key Concepts & Definitions

  • Antacids: Basic substances that neutralize gastric acid, providing symptomatic relief in acid-related disorders.
  • Neutralization Reaction: The chemical process where antacids react with hydrochloric acid (HCl) to produce water and salt, increasing gastric pH.
  • Gastric pH: The measure of acidity in the stomach; normal gastric pH ranges from 1.5 to 3.5.
  • Acid Rebound: A phenomenon where gastric acid secretion increases after the effect of antacids wears off, potentially worsening symptoms.
  • Rapid Onset: Antacids act quickly, usually within minutes, to neutralize existing stomach acid.
  • Duration of Action: The period during which antacids maintain elevated gastric pH, typically 1-2 hours.

Essential Points

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Quiz preview

1. What is the mechanism of action of antacids?

2. What is the primary mechanism by which antacids provide relief from acid-related disorders?

3. Which of the following is a type of antacid that contains aluminum hydroxide?

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Flashcards preview

Antacids — mechanism?

Neutralize gastric acid chemically.

Antacids — mechanism?

Neutralize gastric acid via chemical reaction.

PPIs — main target?

H+/K+ ATPase enzyme in parietal cells.

Gastric pH — normal range?

1.5 to 3.5

Types of PPIs?

Omeprazole, Esomeprazole, Lansoprazole, Pantoprazole, Rabeprazole.

Acid rebound — cause?

Increased acid secretion after antacid effect wanes.

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